ERCC4 and Friedreich ataxia: The unhooked adduct in the opposite strand is repaired by nucleotide excision repair (NER); FANCQ/XPF protein participates in both FA/BRCA and NER pathways, however FA patients with mutations in this gene, do not share the phenotype with Xeroderma Pigmentosum patients, making evident that is a multitask protein [19].