NDUFS4 and alopecia: There is also other evidence that dysfunctional mitochondrial complex I may contribute to the Aifm1Hq/Y pelage phenotype (Vahsen et al. 2004; Bénit et al. 2008; Kruse et al. 2008), since the alopecia in Aifm1Hq/Y mice resembles the alopecia in mice with functional inactivation of the complex I subunit NDUFS4 (Kruse et al. 2008).