Compared with the control group, the abundance of proautophagy proteins (Beclin1, p62, Atg7, Atg5, and LC3II/LC3I) was reduced in APAP-induced ALI in mice, whereas phosphorylation of mTOR at serine 2,448 was upregulated and the protein abundance of these proautophagy proteins was significantly recovered by CD treatment (Figures 7A–F). The gene discussed is MTOR; the disease is acute respiratory distress syndrome.