MAGL expression is highly elevated in human cancer cells and primary tumors, including PCa, neuroblastoma, HCC, colorectal, ovarian, endometrial cancers, etc. Aggressive cancer cells do indeed acquire the ability to liberate FFAs from neutral lipid stores because of the heightened expression of MAGL, which is the principal regulator of FFA levels (47). The gene discussed is MGLL; the disease is posterior cortical atrophy.