VEGFA and myocardial infarction: Wang et al. found that the expression of exosomal circHIPK3 derived from hypoxia-induced myocardial cells is upregulated, which promotes angiogenesis and limits the area of myocardial infarction, partly via the miR-29a/VEGFA axis, to maintain myocardial function and integrity of ECs after myocardial infarction and protect the myocardium.