CAD patients with impaired renal function showed an increased proportion of intermediate CD14++CD16+ monocytes than patients with CAD and eGFR > 60 mL/min (p = 0.014) suggesting a role for renal function in the generation of inflammatory environments associated with ATH progression (Table 2 shows the distribution of monocytic subpopulations in patients with CAD and CKD). This evidence concerns the gene CD14 and chronic kidney disease.