Ke and colleagues found that the expression of both FABP5 and PPARγ, but not PPARβ/δ, increases in PCa cell lines compared to benign cell lines and prostatic hyperplasia tissues, and that the cytoplasmic levels of FABP5 correlate with nuclear immunointensity of PPARγ in PCa tissues [36]. This evidence concerns the gene FABP5 and posterior cortical atrophy.