In addition, anti-ganglioside antibodies tend to be more closely associated with the axonal subtype of GBS than the demyelination subtype, and plasma cells are the indispensable source of these antibodies.[3,42] Simultaneously, plasma cells are also the main producers of IL-36α and IL-36γ, which may also explain the evaluated levels of IL-36α and IL-36γ in the axonal subtype of GBS. This evidence concerns the gene IL36A and Guillain-Barre syndrome.