IL36A and Guillain-Barre syndrome: GBS is an autoimmune neurological disorder characterized by limb weakness, weakening, or disappearance of tendon reflexes and progression over several weeks followed by gradual recovery.[2] Our results showed that serum IL-36α and IL-36γ levels were significantly elevated in patients with GBS during the acute phase compared to the HC group, and these cytokines tended to decrease in the recovery phase.