The CSF levels of IL-36α and IL-36γ in patients with the axonal subtype of GBS were higher than those in patients with the demyelination subtype (P < .05, Fig. 6e and g), while significant differences in CSF IL-36β and IL-36Ra levels were not observed between patients with different clinical subtypes of GBS (P > .05, Fig. 6f and h). This evidence concerns the gene IL36G and Guillain-Barre syndrome.