Many pathogenic antibodies and associated immune cells play crucial roles in the occurrence and development of GBS, including plasma cells and plasmacytoid dendritic cells.[39,40] Plasma cells, monocytes, and dendritic cells are the main producers of IL-36 cytokines, particularly IL-36α and IL-36γ, which may partially explain the different changes of IL-36α, IL-36γ, and IL-36β in GBS.[35,36] Additionally, similar results were observed in CSF in patients with GBS. Here, IL36B is linked to Guillain-Barre syndrome.