The higher prevalence and poorer control of hypertriglyceridemia in KTR were probablyrelated to side effects of immunosuppressive drugs, such as prednisone, calcineurininhibitors, and mammalian-target of rapamycin inhibitors (m-TORi).22 Although untreated hypercholesterolemia wasless often observed in KTR, concerns regarding avoidance of polypharmacy,potentially harmful drug interactions, and adverse drug effects may have preventedthe use of fibrates in KTR. The gene discussed is MTOR; the disease is hypertriglyceridemia.