IL1B and acute respiratory distress syndrome: B4 significantly recovered mice's lung function and decreased LYMPH (Figure 5(b)), NEUT (Figure 5(c)), WBC (Figure 5(d)), TNF-α (Figure 5(e)), IL-6 (Figure 5(f)), IL-1β (Figure 5(g)), and myeloperoxidase (MPO) (Figure 5(h)) in LPS-induced ALI mouse models.