Our flow cytometric analysis revealed an increase of EPCs (CD45–CD31+CD34+), hemangiogenic precursors (CD45–CD117+CD309+), epithelial precursors (CD45–CD31–Sca1+CD49f+), and differentiating SSCs in the testes of mice with MD treated with BDDA compared to group 2 (Figure 5 and Table 3). This evidence concerns the gene PECAM1 and Menkes disease.