According to this, Netea et al. (57) observed that TLR-2 deficient mice were more resistant to disseminated candidiasis than WT, as there was increased chemotaxis and enhanced candidacidal capacity of TLR-2-/- macrophages due to a more robust Dectin-1-mediated immune response in the absence of TLR-2. This evidence concerns the gene TLR2 and disseminated candidiasis.