C1QTNF3 and myocardial infarction: MSCs with the overexpression of CTRP3 potentiates the protective effects of MSCs on cardiac post-MI via improving pathologic remodeling, and promoting angiogenesis, possibly involving the activation of ERK1/2 and the up-regulation of MMP9 and MT1/2/SOD2 (Zhang Z. et al., 2019).