The Western blotting technique of protein analysis in rats suffering from daunorubicin-induced kidney damage has revealed upregulation of PPARγ and attenuation of AT1R, ETAR, p-NFκB p65, and p-ERK1/2 signaling pathways, thereby reducing oxidative stress and inflammation following naringenin treatment. The gene discussed is AGTR1; the disease is Nephropathy.