However, animals ablated for both Yap and Taz in adult myelinating SCs and oligodendrocytes exhibit severe weight loss, tremors, ataxia, and mortality within 2 weeks (Figure S1) (Deng et al., 2017; Grove et al., 2017), which prevents the assessment of the long‐term effect of YAP and TAZ loss required to make conclusions on their role in myelin maintenance and nerve regeneration after injury. The gene discussed is WWTR1; the disease is cerebellar ataxia.