AKT1 and neoplasm: β-Sitosterol-D-glucoside can inhibit the growth and lung metastasis of animal tumors to some extent, upregulate miR-10a expression, inhibit PI3K-AKT signaling pathway, reverse the expression levels of EMT, and inhibit the expression of metastatic related cytokines, eventually inducing apoptosis and inhibit metastasis in tumor cells [45].