2018) and eventually causes chronic kidney damage by inducing an inflammatory response (Carney 2018) because high concentrations of ALB can cause damage to renal tubular epithelial cells and glomerular podocytes (Simpson and Shand 1983; Jarad et al. 2016; Xiao et al. 2016). Thus, urinary ALB excretion is often used as an indicator of acute kidney injury (Bolisetty and Agarwal 2011). This evidence concerns the gene ALB and acute kidney injury.