Promotes cell cycle arrest via hyperacetylation of tubulin, p53, Foxo3a, and KU70; reduced xenograft tumor growth in a mouse Burkitt lymphoma model; reduced neuroblastoma formation in N-Myc transgenic mice; and Repressed aromatase transcription via ERα deacetylation in breast cancerDecreased proinflammatory cytokines expression and macrophage response upon microbial stimulation (TLR) in vivo. This evidence concerns the gene CYP19A1 and Burkitt lymphoma.