ACADVL and medium chain acyl-CoA dehydrogenase deficiency: Mounting evidence of altered mitochondrial morphology, functions, and dynamics has been recently described in various tissues of patients with MCAD, LCHAD/MTP, and VLCAD deficiencies and in the genetic models of these diseases, suggesting that impairment of mitochondrial homeostasis may play a relevant role in the pathogenesis of these disorders.