Considering that OM-peptides induce antigen-specific prophylactic and therapeutic T cell tolerance across different mouse/human MHC class II in EAE models, and with view to moving studies closer to MOGAD and possibly MS, we investigated the frequencies of proliferation responses to candidate myelin autoantigens across HLA-DRB1 genotypes, in PBMC from Greek MS patients and healthy individuals (Table 1). The gene discussed is HLA-DRB1; the disease is myeloid sarcoma.