MOG and Autoimmunity: This notion is also supported by EAE animal experiments, where uninfected B-cells prevented autoimmunity by degrading self-antigens, while these antigens, which are generated by the productive processing of myelin oligodendrocyte glycoprotein (MOG), are presented to autoreactive T-cells in EBV infected B-cells, thereby inducing an immune activity (Thorley-Lawson and Mann, 1985; Livingston et al., 1997; Jagessar et al., 2016; Dunham et al., 2017; Jakimovski et al., 2017; Morandi et al., 2017; Guan et al., 2019).