Beside the classic antineuronal antibodies causing autoimmune encephalitis several antibodies targeting proteins adjacent to the node of Ranvier (paranodal), such as contactin-1 (CNTN1), neurofascin-155 (NF155), nodal neurofascin (NF140/186) and contactin-associated-protein-1 (CASPR1) were described in patients who developed a disabling tremor in the context of CIDP [77, 78]. This evidence concerns the gene CNTN1 and chronic inflammatory demyelinating polyradiculoneuropathy.