HDAC6 modulates the stress response, oncogenesis, cell motility, and many other cancer-related signaling networks in ovarian cancer cells [10,24], and its inhibition significantly improved the survival of mice bearing AT-Rich Interaction Domain 1A (ARID1A)-mutated ovarian tumors, correlating with suppressed tumor growth and dissemination of ARID1A-mutated, but not wild-type, tumors [25]. This evidence concerns the gene ARID1A and neoplasm.