Other temporin isoforms, i.e., TA and TB, have already been characterized for their plausible development as anti-infective agents against S. aureus-induced skin infections; they were found to kill S. aureus internalized into HaCaT keratinocytes and to stimulate migration of these cells by the epidermal growth factor receptor-mediated signaling pathway [60]. This evidence concerns the gene EGFR and tuberculosis.