B6 (expressing iNKT and II NKT cells) and Jα18-/- mice (expressing only type II NKT cells) had similar bacterial burdens in the liver and kidney at all timepoints tested post infection, while CD1d-/- mice (lacking both NKT cell subsets) again had significantly increased bacterial burdens in the liver and kidney at 4 dpi (Figures 2A, B), which suggested that type II NKT cells were sufficient to control SA growth in the absence of iNKT cells at this early timepoint. Here, CD1D is linked to infection.