EGFR and nasopharyngeal carcinoma: In parallel with this, intracellular ROS detection showed that both H‐EVs treatment and EGFR overexpression could induce the decrease of ROS levels in 6–10B and S26 cells, which was reversed in these cells with EGFR‐KO H‐EVs treatment or EGFR knockdown (Figure 5d), implying that H‐EVs induced up‐regulation of EGFR reduces ROS levels in NPC cells.