T11TS treatment repressed the NF‐κB signalling pathway in glioma‐induced animal models and thus induced the downregulation of pro‐inflammatory cytokines and upregulation of anti‐inflammatory cytokines, IL‐4 and IL‐10, for glioma angiogenesis elimination68 (Table 1). The gene discussed is NFKB1; the disease is central nervous system cancer.