MALAT1 and myocardial infarction: Another study suggested that MALAT1 may function as a ceRNA to upregulate NLRC5 (nucleotide-binding and oligomerization domain-like receptor C5) by binding to miR-125b-5p in an IRI-induced acute myocardial infarction (AMI) mouse model, leading to the apoptosis of myocardial cells [21].