In contrast, fasting blood glucose and glucose intolerance levels in the GCN2KO HFD+EXE group remained nearly unchanged compared to the GCN2KO HFD group, indicating that GCN2 deficiency did impair the GCN2KO HFD+EXE group's glucose clearance ability, which exercise failed to ameliorate (Figures 1(g) and 1(h)). The gene discussed is EIF2AK4; the disease is glucose measurement.