The higher upregulation of pro-inflammatory genes (Ifnγ, Kc, Inos, Il1β) and the higher concentration of immune proteins (MPO, KC) that we observed in SA+/ST−/mCRAMP−/− and SA+/ST+/mCRAMP−/− mice are clear indications of a higher susceptibility to salmonellosis when cathelicidin is absent. This evidence concerns the gene IL1B and salmonellosis.