FOXP3 and acute respiratory distress syndrome: We previously showed that CD4+CD25+Foxp3+ Tregs resolve experimental ALI by modulating the following critical prorepair steps: (a) abrogation of macrophage proinflammatory responses (29), (b) augmentation of neutrophil efferocytosis (29), (c) limitation of fibroproliferation (30), and (d) augmentation of alveolar epithelial repair (31).