We showed that CD4+CD25+Foxp3+ Tregs resolve experimental ALI by modulating critical prorepair steps: (a) abrogation of macrophage proinflammatory responses, (b) augmentation of neutrophil efferocytosis (29), (c) limitation of fibroproliferation (30), and (d) augmentation of alveolar epithelial repair (31). This evidence concerns the gene IL2RA and acute respiratory distress syndrome.