It has been reported that the cytotoxic action of ALX is mediated by the formation of free radicals such as superoxide and catalase radicals, which selectively damages the β cells of the pancreas, which are known to be one of the weakest structures to oxidative stress by generating excess reactive oxygen species and produces heart lesions that are similar to human diabetic cardiomyopathy [4, 6]. Here, CAT is linked to diabetic cardiomyopathy.