In ovarian carcinoma cell lines, 18βGA evoked apoptosis via potentiation of trichostatin A (1–25 μM, 24 h) (Lee et al., 2010) and ΔΨM depolarisation leading to Hsp90 inhibition‐mediated caspase 8 activation (Yang, Myung, Kim, & Lee, 2012) or cytochrome c release and caspase 3 activation (Lee, Kim, Lee, Han, & Lee, 2008). The gene discussed is CASP8; the disease is ovarian carcinoma.