Thus, several preclinical tumor transplantation studies aimed at increasing the accumulation of tumor-resident cDC1s and noted that NK cell-derived fms like tyrosine kinase 3 ligand (FLT3L) and other cDC1 chemoattractants stimulated cDC1 recruitment to the tumor and controlled further tumor growth16,17. The gene discussed is FLT3LG; the disease is neoplasm.