CCL2 and rosacea: In an in vitro rosacea skin lesion model, in response to the antibacterial peptide LL-37 (cathelicidin) the production of MCP-1 was increased in HaCaT keratinocytes through the activation of JAK2/STAT3 signaling, suggesting that these pathways may play an important role in the inflammation caused by D. folliculorum [205].