BACE1 and amyloidosis: Future investigations may extend the present findings investigating the relationships among the present spectral EEG markers and the amyloid plaque formation, neuroinflammatory processes (e.g., by biomarkers of mRNA or protein expression), and cognitive performances in groups of at least 15 younger TASTPM mice (5 to 9 months of age) of both sexes for longer chronic administrations (i.e., several months) of the BACE-1 inhibitor ER-901356 in relation to vehicle and a control drug condition sensitive to AD neuropathology and EEG biomarkers (e.g., GABA-A agonist muscimol; [73]).