However, vessel endothelium activation by MCs, in line with their capacity to produce the T-cell chemoattractants CCL2 (MCP-1), CCL5 (RANTES), and CXCL10 (IP-10), indicates a contribution to the recruitment of effector T cells, once they are expanded in lymphoid organs, to the peripheral site of inflammation or infection [126,127,128,129]. This evidence concerns the gene CCL5 and infection.