Moreover, ALK not only fuses with EML4 but also forms genetic fusions with other mRNA partners such as STRN, TPM1, PPP4R3B, ACTG2, STK39, KCNQ5, KCNQ5, MALAT1, GTF2IRD1, GALNT14, and SCEL, which, according to data retrieved from the TCGA PanCancer Atlas using cBioPortal (10,953 patients/10,967 samples), contribute to the development of eight other cancer types (Table 1). This evidence concerns the gene EML4 and cancer.