In a model of cardiac hypertrophy induced by AngII administration, a significant increase in HuR cytoplasmic translocation, indicative of its activation, was observed in neonatal rat hypertrophic cardiomyocytes [59], thus emphasizing the involvement of HuR in the AngII-mediated increase of VEGF protein expression, as observed in our model. The gene discussed is AGT; the disease is cardiac hypertrophy.