In the lungs, interaction of different immune active molecules such as interleukins, chemokines, colony-stimulating factor and tumor necrotic factor alpha (TNF-α) ensues thereby triggering proinflammatory response ultimately leading to respiratory distress syndrome (RDS), septic shock and multiple organ failure in severe cases with consequent formation of free radicals [2]. Here, CSF2 is linked to newborn respiratory distress syndrome.