As expected, infection of air-exposed Calu-3 cells with influenza H1N1 significantly induced the release of IFN-α2a (0 vs. 12.3 pg/mL), IFN-λ (220 vs. 19,647 pg/mL), and IP-10 (119 vs. 3,370 pg/mL) compared to uninfected controls. This evidence concerns the gene IFNA2 and influenza.