Other studies supported this PMT in GBM[33, 34, 35] and showed that tafazzin (TAZ), signal transducer and activator of transcription 3 (STAT3), and CCAAT enhancer binding protein beta (C/EBP‐β) are responsible for reprograming PN glioma stem‐like cells (GSCs) into MES subtype, while the nuclear factor kappa‐light‐chain‐enhancer of activated B cells (NF‐kB) act as the main mediator. The gene discussed is CEBPB; the disease is glioma.