In a previous study, we also demonstrated that exogenous upregulation of HOXA9 inhibited tumor cell invasion and migration and attenuated the expression of snail family zinc finger 2 (SNA12/SLUG) through repression of nuclear factor (NF)-kB activity and suggested that HOXA9 can serve as a potential target for developing anticancer agents [10]. The gene discussed is SNAI2; the disease is neoplasm.