CRP and metabolic syndrome: In conclusion, high frequencies of cTfh2 and cTfh17 subsets in high‐stenosis group and their correlation with cholesterol, CRP, ESR, and NLR suggest an ongoing deviation of nonefficient subsets toward efficient phenotype in the context of inflammation and dyslipidemia and also indicate that these cells, as an effector phenotype, may be involved in atherosclerosis‐related immune responses.