Autosomal dominant inherited hypercholesterolemia (ADH) accounts for a major proportion of the FH cases worldwide with mutations most commonly reported in low density lipoprotein receptor (LDLR), apolipoprotein B (APOB), and proprotein convertase subtilisin/kexin type 9 (PCSK9) [3–5]. Here, LDLR is linked to familial hyperaldosteronism.