Therefore, the results on the prediction of targeted activity indicate a high probability of interaction of compound 3 with a large number of biotargets responsible for a decrease in insulin resistance: peroxisome proliferator-activated receptor delta, free fatty acid receptor 1 (GPR40), peroxisome proliferator-activated receptor gamma, free fatty acid receptor 2 (GPR43), protein-tyrosine phosphatase 1B (PTP1B), and free fatty acid receptor 4 (GPR120). This evidence concerns the gene PPARG and Insulin resistance.