We also found that the serum level of IL6 was tightly associated with the biomarker cTnI in all patients with COVID-19 at different time points, which supports the point of view that the inflammation starting in and propagating from the lung or other initial organ injuries probably resulted in some bystander effects on other organs, such as the heart, due to amplifying inflammatory responses [9, 12]. Here, TNNI3 is linked to COVID-19.