Earlier, we reported differences in the metabolic parameters (body weights, adiposity index), biochemical parameters (serum glucose, cholesterol, triglyceride levels), hormonal profiles (serum testosterone, estradiol, LH, FSH, prolactin, leptin, insulin levels), fertility parameters (implantation losses, litter size, potency) and spermatogenesis (mitosis, meiosis, spermiogenesis) between the two types of obesity could be attributed to the difference in the white adipose tissue accumulation despite both the groups having similar body weights [10, 14]. This evidence concerns the gene INS and obesity due to melanocortin 4 receptor deficiency.