In addition to the reported benefits of having the antioxidant activity of anthocyanins to protect against cardiovascular diseases [42,43], these compounds also inhibit rate-limiting enzyme activities for cholesterol synthesis—namely, 3-hydroxy-3-methyl-glutaryl-coenzyme A (HMG-CoA) and cholesteryl ester transfer protein (CEPT) enzymes, which are linked to changes in endogenous cholesterol synthesis [43,44,45]. The gene discussed is CETP; the disease is cardiovascular disorder.