Thus, lymphoma patients homozygous for the FcγRIIIA-158V allelic version, which is bound by the antibody’s Fc region with higher affinity, showed better responses to rituximab therapy than did patients carrying the low-affinity FcγRIIIA-158F allele, suggesting functions as ADCC or ADCP as important mechanisms by which the antibody depletes lymphoma cells [16,17,18]. This evidence concerns the gene FCGR3A and lymphoma.